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Index by author

September 01, 2016; Volume 37,Issue 9

  1. Burns, P.

    1. FELLOWS' JOURNAL CLUBPediatric Neuroimaging
      Correlation of MRI Brain Injury Findings with Neonatal Clinical Factors in Infants with Congenital Diaphragmatic Hernia
      R. Radhakrishnan, S. Merhar, J. Meinzen-Derr, B. Haberman, F.Y. Lim, P. Burns, E. Zorn and B. Kline-Fath
      American Journal of Neuroradiology September 2016, 37 (9) 1745-1751; DOI: https://doi.org/10.3174/ajnr.A4787

      The authors evaluated MRIs performed before hospital discharge in 53 infants with congenital diaphragmatic hernia that were scored forbrain injury by 2 pediatric neuroradiologists. Potential associations with perinatal and clinical variables from the neonatal intensive care unit stay were probed. The most common findings were enlarged extra-axial spaces (36%), intraventricular hemorrhage (23%), ventriculomegaly (19%), white matter injury (17%), and cerebellar hemorrhage (17%). Brain injury score was associated with extracorporeal membrane oxygenation, lack of oral feeding at discharge, use of inotropes, and gastrostomy tube placement before hospital discharge.

  2. Burrows, A.

    1. ADULT BRAIN
      Nonaneurysmal Perimesencephalic Hemorrhage Is Associated with Deep Cerebral Venous Drainage Anomalies: A Systematic Literature Review and Meta-Analysis
      A. Rouchaud, V.T. Lehman, M.H. Murad, A. Burrows, H.J. Cloft, E.P. Lindell, D.F. Kallmes and W. Brinjikji
      American Journal of Neuroradiology September 2016, 37 (9) 1657-1663; DOI: https://doi.org/10.3174/ajnr.A4806

  3. Bush, A.

    1. Pediatric Neuroimaging
      In Vivo T1 of Blood Measurements in Children with Sickle Cell Disease Improve Cerebral Blood Flow Quantification from Arterial Spin-Labeling MRI
      L. Václavů, V. van der Land, D.F.R. Heijtel, M.J.P. van Osch, M.H. Cnossen, C.B.L.M. Majoie, A. Bush, J.C. Wood, K.J. Fijnvandraat, H.J.M.M. Mutsaerts and A.J. Nederveen
      American Journal of Neuroradiology September 2016, 37 (9) 1727-1732; DOI: https://doi.org/10.3174/ajnr.A4793

  4. Calmon, R.

    1. Pediatric Neuroimaging
      Response Assessment in Pediatric Neuro-Oncology: Implementation and Expansion of the RANO Criteria in a Randomized Phase II Trial of Pediatric Patients with Newly Diagnosed High-Grade Gliomas
      T. Jaspan, P.S. Morgan, M. Warmuth-Metz, E. Sanchez Aliaga, D. Warren, R. Calmon, J. Grill, D. Hargrave, J. Garcia and G. Zahlmann
      American Journal of Neuroradiology September 2016, 37 (9) 1581-1587; DOI: https://doi.org/10.3174/ajnr.A4782

  5. Cekirge, S.

    1. EDITOR'S CHOICENeurointervention
      Risk Factors for Ischemic Complications following Pipeline Embolization Device Treatment of Intracranial Aneurysms: Results from the IntrePED Study
      W. Brinjikji, G. Lanzino, H.J. Cloft, A.H. Siddiqui, E. Boccardi, S. Cekirge, D. Fiorella, R. Hanel, P. Jabbour, E. Levy, D. Lopes, P. Lylyk, I. Szikora and D.F. Kallmes
      American Journal of Neuroradiology September 2016, 37 (9) 1673-1678; DOI: https://doi.org/10.3174/ajnr.A4807

      This is a retrospective subanalysis of the IntrePED study, which has beenpreviously published (AJNR Am J Neuroradiol 2015;36:108–15).Seven hundred ninety-three patients with 906 treated aneurysms were enrolled. Thirty-six (4.5%) patients had postoperative acute ischemic stroke, 21 of which occurred within 1 week of the procedure. There was no difference in the rate of acute stroke between the anterior and posterior circulations. Stroke rate was 3% in patients with 1 PED, and 7% in those with 2 PEDs. With multivariate analysis, the only variable independently associated with postoperative stroke was treatment of fusiform aneurysms. Among the patients with stroke, 10 (27.0%) died and 26 (73.0%) had major neurologic morbidity. The authors conclude that acute ischemic stroke following treatment of intracranial aneurysms with the PED is an uncommon but devastating complication, with 100% of patients having major morbidity or mortality.

  6. Chen, W.

    1. EDITOR'S CHOICEADULT BRAIN
      Open Access
      Quantitative Susceptibility Mapping and R2* Measured Changes during White Matter Lesion Development in Multiple Sclerosis: Myelin Breakdown, Myelin Debris Degradation and Removal, and Iron Accumulation
      Y. Zhang, S.A. Gauthier, A. Gupta, W. Chen, J. Comunale, G.C.-Y. Chiang, D. Zhou, G. Askin, W. Zhu, D. Pitt and Y. Wang
      American Journal of Neuroradiology September 2016, 37 (9) 1629-1635; DOI: https://doi.org/10.3174/ajnr.A4825

      The authors characterized lesion changes on quantitative susceptibility mapping and R2* at various gadoliniumenhancementstages (nodular, shell-like, nonenhancing) in 64 patients with 203 lesions. They found that: 1) active MS lesions with nodular enhancement show R2* decrease but no quantitative susceptibility mapping change; 2) late active lesions with peripheral enhancement show R2* decrease and quantitative susceptibility mappingincrease in the lesion center; and 3) nonenhancing lesions show both quantitative susceptibility mapping and R2* increase, reflecting iron accumulation.

  7. Chiang, G.C.-Y.

    1. EDITOR'S CHOICEADULT BRAIN
      Open Access
      Quantitative Susceptibility Mapping and R2* Measured Changes during White Matter Lesion Development in Multiple Sclerosis: Myelin Breakdown, Myelin Debris Degradation and Removal, and Iron Accumulation
      Y. Zhang, S.A. Gauthier, A. Gupta, W. Chen, J. Comunale, G.C.-Y. Chiang, D. Zhou, G. Askin, W. Zhu, D. Pitt and Y. Wang
      American Journal of Neuroradiology September 2016, 37 (9) 1629-1635; DOI: https://doi.org/10.3174/ajnr.A4825

      The authors characterized lesion changes on quantitative susceptibility mapping and R2* at various gadoliniumenhancementstages (nodular, shell-like, nonenhancing) in 64 patients with 203 lesions. They found that: 1) active MS lesions with nodular enhancement show R2* decrease but no quantitative susceptibility mapping change; 2) late active lesions with peripheral enhancement show R2* decrease and quantitative susceptibility mappingincrease in the lesion center; and 3) nonenhancing lesions show both quantitative susceptibility mapping and R2* increase, reflecting iron accumulation.

  8. Cho, H.R.

    1. ADULT BRAIN
      Open Access
      Antiangiogenic Effect of Bevacizumab: Application of Arterial Spin-Labeling Perfusion MR Imaging in a Rat Glioblastoma Model
      T.J. Yun, H.R. Cho, S.H. Choi, H. Kim, J.-K. Won, S.-W. Park, J.-h. Kim, C.-H. Sohn and M.H. Han
      American Journal of Neuroradiology September 2016, 37 (9) 1650-1656; DOI: https://doi.org/10.3174/ajnr.A4800

  9. Choi, S.H.

    1. ADULT BRAIN
      Open Access
      Antiangiogenic Effect of Bevacizumab: Application of Arterial Spin-Labeling Perfusion MR Imaging in a Rat Glioblastoma Model
      T.J. Yun, H.R. Cho, S.H. Choi, H. Kim, J.-K. Won, S.-W. Park, J.-h. Kim, C.-H. Sohn and M.H. Han
      American Journal of Neuroradiology September 2016, 37 (9) 1650-1656; DOI: https://doi.org/10.3174/ajnr.A4800

  10. Claassen, J.

    1. ADULT BRAIN
      Open Access
      Application of Blood-Brain Barrier Permeability Imaging in Global Cerebral Edema
      J. Ivanidze, O.N. Kallas, A. Gupta, E. Weidman, H. Baradaran, D. Mir, A. Giambrone, A.Z. Segal, J. Claassen and P.C. Sanelli
      American Journal of Neuroradiology September 2016, 37 (9) 1599-1603; DOI: https://doi.org/10.3174/ajnr.A4784
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