Article Figures & Data
Figures
- Fig 1.
Tracking results of the CSTs (yellow) and the TRS1s (blue) in a healthy control (column 1) and 3 subjects with unilateral CP (columns 2–4, On-line Table). Images, shown in radiologic orientation, include T1-weighted images (row 1) and tracts superimposed on FA maps in the axial section at the level of the upper corona radiata (row 2) and entire coronal projections (rows 3–4). In the control (column 1), the CSTs are initiated from the entire M1 ROIs and follow consistent, expected paths down to the brain stem. The TRS1s are primarily initiated in the ventroposterior parts of the thalami, then travel posterior to the CSTs in the corona radiata, and spread over a large part of the somatosensory cortex ROIs. Subject 9 (column 2) has a periventricular pseudocyst at the level of corona radiata, which causes local splits to the left TRS1 and left CST, which are seen to partly share locations, and dislocate the major branch of the left CST. Subject 12 (column 3) has a large right WM lesion obstructing the anticipated courses of the TRS1s and the CSTs, making tracking of the TRS1 impossible and dislocating the right CST. Subject 14 (column 4) had a left focal infarct with extensive damage to the entire left hemicranium and severe WM damage above the PLIC, which confined the left CST to a thin sliver of WM at the level of the corona radiata and made the left TRS1 nontrackable.
- Fig 2.
Boxplots of diffusion parameters in the TRS1, CST-M1 to PLIC, and CST-PLIC to CerPed in the group with unilateral CP and controls on the lesion (gray) and nonlesion (white) sides. Diffusivity is expressed as 10−4 mm2/s. The asterisk indicates a significant difference between the group with unilateral CP and controls by statistical comparisons using the Mann-Whitney U test (2-tailed; significance level, .05).
