Article Figures & Data
Figures
- Fig 1.
TSE (A, D), TGSE (B, E), and TIDE (C, F) images of a 38-year-old healthy man. The results show similar T2 contrast of the neocortex (A–C). At the basal ganglia, iron-containing gray matter (eg, globus pallidus) has a much higher intensity in the TIDE image compared with the other 2 results (D–F). Ghosting artifacts appear in the TIDE image (arrow in C).
- Fig 2.
TSE (A, D), TGSE (B, E), and TIDE (C, F) images of a 25-year-old healthy man. Motion artifacts can be found in the TSE and TGSE images, due to eyeball movement (open arrows in A and B) and CSF flow (open arrows in D and E). Magnetic susceptibility artifacts appear in the TIDE image (solid arrows in C). Occasional basilar artery flow artifacts are seen in the TIDE image (thin arrow in C).
- Fig 3.
TSE (A, D), TGSE (B, E), and TIDE (C, F) images of a 57-year-old woman with stroke history. A hyperintense lesion appears near the left lateral ventricle (white arrows in A–C). The degrees of hyperintensity are different in these images. Similar contrast of a late subacute hematoma involving the right corpus striatum is found in all sequences (black arrows in D–F).
- Fig 4.
A 9-day-old neonate with seizures, presenting with involuntary movements, was imagined with TSE (A) and TIDE (B) sequences. A motion artifacts-free T2-weighted image can be acquired with TIDE, depicting a clear brain anatomy.
- Fig 5.
TSE (A), TGSE (B), and TIDE (C) images of a 26-year-old female volunteer. CSF motion artifacts (double arrows) are seen on TSE and TGSE images, while eyeball motion artifacts (open arrows) are seen on the TGSE image. The trigeminal nerves (arrows) are clearly demonstrated on TIDE, which is free of the aforementioned motion artifacts but are somewhat blurred on TSE and TGSE.
- Fig 6.
TSE (A, D), TGSE (B, E), and TIDE (C, F) images of a 40-year-old male volunteer at the levels of the centrum semiovale (star, A–C) and thalamus (triangle, D–F). The gray-white matter junctions (double arrows) are clearly demonstrated on TSE but are blurred on TGSE and TIDE.
Tables
SNRs and CNRs of TSE, TGSE, and TIDE sequences
TSE TGSE TIDE PTSE,TGSE PTGSE,TIDE PTSE,TIDE SNR WM CSO 56.14 ± 8.05 29.29 ± 4.36 25.02 ± 3.70 – – – CR 56.48 ± 8.05 29.71 ± 4.28 25.86 ± 3.63 – – – aPVWM 52.54 ± 6.94 27.35 ± 3.57 24.12 ± 3.25 – – – pPVWM 58.58 ± 7.48 31.05 ± 3.85 28.20 ± 3.59 – – – GM CH 68.38 ± 9.35 35.80 ± 4.69 32.39 ± 4.34 – – – PUT 61.97 ± 8.52 32.11 ± 4.25 32.58 ± 4.35 – .364 – THA 61.09 ± 8.18 32.03 ± 4.18 30.71 ± 3.76 – .031 – GP 48.47 ± 6.58 24.80 ± 3.42 28.30 ± 3.95 – – – CNR NICGM-WM, THA-CSO 4.95 ± 3.64 2.74 ± 1.85 5.69 ± 1.37 – – .169 ICGM-WM, GP-CSO −7.67 ± 3.98 −4.494 ± 2.35 3.275 ± 1.70 – – – Note:—CSO indicates centrum semiovale; CR, corona radiata; aPVWM, anterior periventricular white matter; pPVWM, posterior periventricular white matter; CH, head of caudate nucleus; PUT, putamen; THA: thalamus; GP, globus pallidus; NICGM, non-iron-containing gray matter; ICGM, iron-containing gray matter; –, P < .0001.
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