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Research ArticleBRAIN

Clinical and Imaging Outcomes after Stroke with Normal Angiograms

Andrew P. Slivka, Greg A. Christoforidis, Eric C. Bourekas, Philip E. Calendine and Margaret A. Notestine
American Journal of Neuroradiology February 2005, 26 (2) 242-245;
Andrew P. Slivka
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Greg A. Christoforidis
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Eric C. Bourekas
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Philip E. Calendine
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Margaret A. Notestine
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    TABLE 1:

    Stroke etiologies by TOAST classification

    EtiologyNumberComment
    Cardioembolic7*5 high-risk sources, 2 medium-risk sources
    Small-vessel occlusion2Possible
    Unknown2Probable
    Other1Postcoronary catheterization
    Multiple9Small-vessel occlusion and cardioembolic (1 high-risk source, 8 medium-risk sources)
    • * Cardiac sources included: atrial fibrillation (n = 3), left ventricular thrombus (n = 1), akinetic left ventricular segment (n = 1), nonbacterial thrombotic endocarditis (n = 1), and atrial septal aneurysm with patent foramen ovale (n = 1).

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    TABLE 2:

    CT and MR imaging findings

    FindingNumber
    No new infarct6
    New abnormality15
        Cortical infarct7
            Single5
            Single with subcortical1
            Multiple1
        Subcortical infarct8
            Single, ≤1.5 cm5
            Single, >1.5 cm2
            Multiple1
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American Journal of Neuroradiology: 26 (2)
American Journal of Neuroradiology
Vol. 26, Issue 2
1 Feb 2005
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Cite this article
Andrew P. Slivka, Greg A. Christoforidis, Eric C. Bourekas, Philip E. Calendine, Margaret A. Notestine
Clinical and Imaging Outcomes after Stroke with Normal Angiograms
American Journal of Neuroradiology Feb 2005, 26 (2) 242-245;

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Clinical and Imaging Outcomes after Stroke with Normal Angiograms
Andrew P. Slivka, Greg A. Christoforidis, Eric C. Bourekas, Philip E. Calendine, Margaret A. Notestine
American Journal of Neuroradiology Feb 2005, 26 (2) 242-245;
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